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ASTRO Annual Meeting 2024

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Challenges SOC

SUPREMO

ForPost-mastectomy breast, node-negative high-risk or 1-3 positive nodes

TL;DREditorial critique: chest-wall-only RT cut 10yr CW recurrence 1.1% vs 2.5% (HR 0.45) but RNI was prohibited.

Why it mattersRadiation oncology

The RT-relevant number is buried in SUPREMO's supplement: node-positive LRR 3.3% vs 4.8%, HR 0.51 (0.27-0.96), significant even with chest-wall-only fields and RNI prohibited. With supraclavicular coverage in 12% and IMN in under 2%, this trial never tested comprehensive PMRT, so it cannot settle the elective nodal decision.

Monday clinic

In a post-mastectomy patient with 1-3 positive nodes staged by SLNB alone, this argues SUPREMO does not license PMRT omission; it says nothing about node-negative pts without adverse features, where EBCTCG also found no benefit.

The longer read

Also covered Jul 7

9 details

ASO Perspectives editorial in Annals of Surgical Oncology, not new trial data. Two radiation oncologists re-read SUPREMO against the PMRT and RNI evidence base. PMRT after neoadjuvant therapy is explicitly out of scope.

The central claim is that SUPREMO tested chest wall alone, not PMRT: RNI was prohibited, supraclavicular nodes were covered in 12% of PMRT-arm pts (n=97), and internal mammary irradiation occurred in fewer than 2% across both arms. Non-UK centres could give RNI in the observation arm, and 12 control pts received supraclavicular RNI.

Only 25% were truly node-negative and would not be offered PMRT under current guidelines; the majority had N1 disease (1-3 nodes). 65% were hormone-receptor positive, TNBC was 10%, and only 14% had SLNB alone with the majority undergoing ALND.

SUPREMO's 10-year chest-wall recurrence fell from 2.5% to 1.1% (HR 0.45) with no gain in overall LRR, DFS or OS. The supplement carries the signal the headline drops: LRR 4.8% to 3.3%, HR 0.51 (95% CI 0.27-0.96) in node-positive pts, not in node-negative.

Approach5yr lymphedema risk
SLNB alone8%
SLNB + RNI11%
ALND alone25%
ALND + RNI30%

EBCTCG 2014 found PMRT cut 10-year LRR by 17.9% and 20-year breast cancer mortality by 8% in 1-3 node-positive women, persisting with a single positive node, with no node-negative benefit. MA-20 and EORTC 22922 both showed RNI benefit in 1-3 node-positive disease despite near-universal ALND, and a later EBCTCG RNI meta-analysis showed gains at 15 years including in contemporary systemic-therapy trials.

the omission question in post-mastectomy pts with 1-3 positive nodes or high-risk node-negative disease staged largely by ALND
Does not represent pts staged by SLNB alone, pts with genomic risk scores, or the post-neoadjuvant setting.

This is a single-perspective editorial from two radiation oncologists, so the framing selects evidence favouring comprehensive RNI, and the reader gets no independent re-analysis of SUPREMO's data. Its strongest number, the node-positive LRR HR 0.51, comes from a supplementary subgroup the trial did not power for.

The authors leave the genuinely open questions to trials in progress: MA.39 (Tailor-RT) for RNI omission at low recurrence score, T-Rex for RNI omission in hormone-sensitive disease with one to two macrometastatic SLNs. They also note the surgical corollary, that ALND should not be chosen to earn a PMRT omission, since ALND is the dominant lymphedema driver.

Editorial, no new data; contests the omission reading of SUPREMO on field design and population grounds, aligning with ASTRO 2025 and NCCN rather than the trial's public messaging.

  • RNI omission in 1-3 node-positive pts staged by SLNB alone
  • Whether low Oncotype score permits comprehensive RNI omission
  • PMRT effect in triple-negative disease after mastectomy
📚 Sources · 📄 1 paper
📄 PAPER Naoum, George E.; Taghian, Alphonse G. · Annals of Surgical Oncology (2026-03)
When Postmastectomy Radiotherapy (PMRT) is not Really PMRT: A Critical Evaluation of SUPREMO Trial
📝 NOTE: add as a criticism of SUPREMO

ASTRO 2024: SBRT for Unfavorable Intermediate-Risk Prostate Cancer

TL;DRConference education session on SBRT for UIR prostate: 5 fractions over 1-2wks, 1-2% bothersome toxicity vs 10-30% with 45-fraction 2D era.

Reported via UroToday →

Trials discussed

RTOG 9408PACE BHYPO-RT-PCFLAME 2.0FORT

Why it mattersRadiation oncology

The actionable detail is the urethral-constraint critique of PACE-B: contouring was optional and constrained only "if visualized" (V44Gy <20%), so hotspots ≥120% put ~48Gy (≈121Gy EQD2) on the urethra. That reframes the 5.4% vs 3.7% G2+ GU gap as a planning artifact, not an SBRT property, and argues for contouring and constraining the urethra in 5-fraction prostate plans.

The longer read
14 details 4 trials watching

ASTRO 2024 education session (EDU 16), not a trial report. Dr Daniel Spratt reviews risk stratification, fractionation history, and the SBRT evidence base for unfavorable intermediate-risk prostate cancer.

Unfavorable intermediate-risk disease, defined since 2013 by Gleason grade group 3 (HR 3.49 for distant mets) or ≥2 intermediate risk factors (HR 2.40). FIR and UIR also separate on cumulative PCSM incidence (p=0.013).

Options span brachytherapy and EBRT (protons or photons/IGRT) across conventional (~40fx), moderate hypofractionation (~20fx), and ultra-hypofractionation (~5fx). Era contrast: 1980s 2D delivered 45 fractions over 9 weeks with 10-30% bothersome GU/GI toxicity; modern SBRT is 5 fractions over 1-2 weeks with 1-2%.

RTOG 9408 secondary analysis anchors the ADT question: in UIR, 4mo ADT improved distant metastasis (HR 0.48, 0.28-0.83, P=.008) and PCSM (HR 0.40, 0.26-0.60, P<.001), with no benefit in FIR. PACE-B 5yr showed no significant EFS difference for SBRT vs conventional/moderate hypofractionation.

QuestionEndpointResult
UIR vs FIR prognosisDistant metastasisHR 2.36 (95% CI 1.44-3.89), P=.001
UIR vs FIR prognosisPCSMHR 1.84 (95% CI 1.29-2.62), P=.001
ADT benefit in UIRDistant metastasisHR 0.48 (95% CI 0.28-0.83), P=.008
ADT benefit in UIRPCSMHR 0.40 (95% CI 0.26-0.60), P<.001
ADT benefit in FIRDM / PCSMNo improvement

PACE-B G2+ GU 5.4% SBRT vs 3.7% control (p=0.28), no significant bowel difference. Pooled SBRT series: late grade ≥3 GU 2.0% (1.4-2.8%) and GI 1.1% (0.6-2.0%), with dose associated with both better biochemical control (P=.018) and worse late G3+ GU (P=.014).

The session's argument is that SBRT toxicity is a planning problem, not a modality problem: PACE-B did not require urethral contouring, so likely hotspots of ≥120% (≥48Gy, ~121Gy EQD2 to urethra) can explain the GU excess. Focal-boost work (36.25Gy/5fx + DIL to 45-50Gy; Loblaw's 35Gy prostate / 25Gy pelvis / 50Gy DIL) points the field toward whole-gland de-escalation with a boost.

Single-speaker synthesis with the speaker's own interpretive framing rather than a systematic review; the urethral-hotspot explanation for PACE-B GU toxicity is inference, not a reported dosimetric analysis. The captured excerpt truncates mid-FLAME 2.0 and never reaches the FORT trial named in the keywords.

unfavorable intermediate-risk prostate cancer treated with definitive external beam RT
Does not represent favorable intermediate-risk disease, where the same RTOG 9408 analysis showed no ADT benefit, nor high-risk or node-positive disease.
📚 Sources · 📄 1 paper
📄 PAPER · UroToday
ASTRO 2024: SBRT for Unfavorable Intermediate-Risk Prostate Cancer
Abstract
ASTRO 2024, Prostate Cancer, stereotactic body radiation therapy (SBRT), external beam radiation therapy (EBRT), image-guided radiation therapy (IGRT), RTOG 9408 (NCT00002597) trial, PACE B trial, FLAME 2.0 trial, FORT trial.
📝 https://www.urotoday.com/conference-highlights/astro-2024/astro-2024-prostate-cancer/155301-astro-2024-sbrt-for-unfavorable-intermediate-risk-prostate-cancer.html
Confirmatory

HYDRA

ForLocalised prostate cancer, definitive external-beam RT

TL;DRNo PFS difference for either isodose (HR 0.92) or dose-escalated MHFRT (HR 0.94), but escalation raises late G2+ GI (OR 1.48).

Why it mattersRadiation oncology

The split that matters is isodose vs dose-escalated MHFRT, not hypofractionation itself: escalation adds no PFS (HR 0.94, 0.82-1.09) and costs bowel on both physician grading (OR 1.48) and patient report (OR 1.68). GU was unchanged in both strata. The schedule decision lands on 60 Gy in 20 fractions.

Monday clinic

In a man with localised prostate cancer starting definitive prostate-only EBRT, this supports an isodose moderately hypofractionated schedule over a dose-escalated one; it does not extend to five-fraction ultrahypofractionation, post-prostatectomy salvage, or whole-pelvis treatment.

The longer read
7 details 5 trials watching

IPD meta-analysis of randomised phase 3 CFRT vs MHFRT trials via the MARCAP consortium. Searches on Dec 15, 2023 and re-run Jan 8, 2025 screened 1696 records down to 7 eligible trials. Three separate analyses: efficacy, physician-scored late toxicity, and patient-reported outcomes.

Localised prostate cancer on trials that published patient-level efficacy AND late toxicity data. 3454 pts across three isodose trials, 2426 pts across four dose-escalated trials. Trials whose CFRT arm fell below modern dose were excluded.

The intervention split is the whole point: isodose MHFRT (same equivalent dose in fewer fractions, eg 60 Gy in 20 fractions) versus dose-escalated MHFRT. The CFRT comparator had to deliver ≥70 Gy in 2 Gy equivalents.

Primary (efficacy): progression-free survival. Co-primary toxicity endpoints: late grade 2 or higher GU and GI. Co-primary PRO endpoints: clinically-significant decrement in urinary or bowel quality of life.

The GI signal sits entirely in the dose-escalated stratum and shows up on both physician grading and patient report; the isodose stratum carries neither. GU odds ran above 1 in both comparisons with intervals crossing unity.

CHHiP and PROFIT established 60 Gy in 20 fractions as non-inferior to conventional fractionation. The escalated schedules were built on the premise that a higher equivalent dose in fewer fractions would improve control; pooled here that premise fails on PFS while adding bowel toxicity.

localised prostate cancer treated with definitive external-beam RT on randomised trials against modern-dose CFRT
Does not represent five-fraction ultrahypofractionation, post-prostatectomy salvage, or whole-pelvis nodal treatment, none of which were tested.

Toxicity scales and PRO instruments were not uniform across the seven trials, and the dose-escalated stratum pools four schedules that are not interchangeable, so the OR describes escalation as a class, not one regimen. Follow-up also differs between strata (5.4 vs 7.1 yrs).

With efficacy answered as a null, the schedule decision turns entirely on toxicity, and the toxicity difference runs one way. Escalating per-fraction dose beyond isodose buys no measurable PFS while adding bowel morbidity that pts themselves report, which leaves little argument for an escalated MHFRT schedule in intact localised disease.

Pooled IPD from seven randomised phase 3 trials, aligned with existing moderate-hypofractionation practice; refines which regimen rather than establishing a new modality or population.

📚 Sources · 📄 1 paper
📄 PAPER Kishan; Sun; Tree et al. · The Lancet. Oncology (2025-04)
Hypofractionated radiotherapy for prostate cancer (HYDRA): an individual patient data meta-analysis of randomised trials in the MARCAP consortium.
Abstract
BACKGROUND: Trials comparing moderately hypofractionated radiotherapy (MHFRT) to conventionally-fractionated radiotherapy (CFRT) for prostate cancer have varied considerably in intent (non-inferiority vs superiority) and MHFRT dose. We compare the efficacy and toxicity profiles of isodose MHFRT and dose-escalated MHFRT.<br/><br/>METHODS: This was an individual patient data meta-analysis that identified randomised phase 3 trials of CFRT versus MHFRT that had published individual patient-level data on efficacy and late toxicity. A systematic literature search using MEDLINE, Embase, trial registries, the Web of Science, Scopus, and relevant conference proceedings was initially conducted on Dec 15, 2023, and was re-conducted on Jan 8, 2025. Trials that did not publish efficacy data, did not publish late toxicity data, or did not use modern dose radiotherapy (&#x2265;70 Gy in 2 Gy equivalents) in the CFRT group were excluded. Individual patient data were provided to MARCAP by study investigators. Three separate meta-analyses were designed to compare efficacy (primary endpoint was progression-free survival), physician-scored late toxicity (co-primary endpoints were late grade 2 or higher genitourinary and late grade 2 or higher gastrointestinal toxic effects), and patient-reported outcomes (co-primary endpoints were clinically-significant decrements in patient-reported urinary or bowel quality of life) between patients receiving CFRT versus MHFRT.<br/><br/>FINDINGS: We identified 1696 records for review. Seven phase 3 trials comparing MHFRT with CFRT were eligible for inclusion in our analysis. Individual patient data were obtained from these seven studies (3454 patients from three trials comparing CFRT with isodose MHFRT and 2426 patients from four trials comparing CFRT with dose-escalated MHFRT). At a median follow-up of 5&#xb7;4 years (IQR 4&#xb7;6-7&#xb7;2) for isodose MHFRT and 7&#xb7;1 years (5&#xb7;7-8&#xb7;4) for dose-escalated MHFRT, no differences in progression-free survival were detected (hazard ratio 0&#xb7;92, 95% CI 0&#xb7;81-1&#xb7;05; p=0&#xb7;21 and 0&#xb7;94, 0&#xb7;82-1&#xb7;09; p=0&#xb7;43 respectively). No increased odds of grade 2 or higher genitourinary toxic effects were identified for either isodose (odds ratio [OR] 1&#xb7;16, 95 CI% 0&#xb7;86-1&#xb7;57; p=0&#xb7;32) or dose-escalated MHFRT (1&#xb7;20, 0&#xb7;95-1&#xb7;51; p=0&#xb7;13). The odds of grade 2 or higher gastrointestinal toxic effects were significantly higher with dose-escalated (OR 1&#xb7;48, 95% CI 1&#xb7;14-1&#xb7;92; p=0&#xb7;0035) but not isodose MHFRT (1&#xb7;30, 0&#xb7;59-2&#xb7;87; p=0&#xb7;51). Isodose MHFRT was not found to show different odds of urinary quality-of-life decrement (OR 1&#xb7;03, 95% CI 0&#xb7;51-2&#xb7;09; p=0&#xb7;93) or bowel quality-of-life decrement (0&#xb7;76, 0&#xb7;40-1&#xb7;43; p=0&#xb7;39). Dose-escalated MHFRT was associated with greater odds of bowel quality-of-life decrement (OR 1&#xb7;68, 95% CI 1&#xb7;07-2&#xb7;61; p=0&#xb7;023), but no evidence of greater urinary quality-of-life decrement was found (1&#xb7;57, 0&#xb7;87-2&#xb7;85; p=0&#xb7;13).<br/><br/>INTERPRETATION: Isodose MHFRT and dose-escalated MHFRT both have similar efficacy compared with CFRT, but dose-escalated MHFRT is associated with higher physician-scored and patient-reported bowel toxicity. Isodose regimens, eg, 60 Gy in 20 fractions, should be the standard MHFRT regimen for localised prostate cancer.<br/><br/>FUNDING: None.